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Acute myocardial infarction answers (182)

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Q: 

What is the pathophysiology of myocardial infarction?

A: The pathophysiology of acute myocardial infarction is complex. Loss of viable myocardium impairs global cardiac function, which can lead to reduced cardiac output, and if damage is severe, to cardiogenic shock. Systolic and diastolic dysfunction are associated with ischemic myocardium. If left ventricular function is significantly impaired, pulmonary congestion and edema can occur. Ischemia can also precipitate abnormal cardiac rhythms and conduction blocks that can further impair function and become life-threatening in some cases. Reduced cardiac output and arterial pressure can elicit baroreceptor reflexes that lead to activation of neurohumoral compensatory mechanisms (e.g., activation of sympathetic nerves and...


Q: 

What is a myocardial infarction?

A: ANSWER: A heart attack. Also called an AMI. An acute myocardial infarction. Or sudden onset. It means that the heart is deprived of oxygen, because of lessenned or lack of bloodflow to the heart....


Q: 

How is ESR affected by myocardial infarction?

A: Answer The ESR (or sedimentation rate) is a marker for acute inflammation. In the case of a myocardial infarction it is typically elevated within 3 days of the infarction and may remain elevated for weeks after. Answer The ESR (or sedimentation rate) is a marker for acute inflammation. In the case of a myocardial infarction it is typically elevated within 3 days of the infarction and may remain elevated for weeks after....


Q: 

Antipsychotics may be Alzheimer''s patients with myocardial infarction

A: @yoxi5236, The incidence of strokes, and death, is also increased when anti-psychotics, particularly atypical anti-psychotics, are prescribed for elderly dementia patients. Many of these drugs carry a black box warning label regarding use for this group. Quote:Despite existing security warning, antipsychotic drugs are still widely prescribed That''s because management issues may over-ride other concerns and risks. It is certainly preferable to find other methods, and try other drugs, before resorting to the use of those with high risk of severely debilitating, and possibly fatal, side-effects. These medications should be used, if at all, as a last resort. @firefly, Management issues meaning it''s less work to care for the patients? Sounds selfish at first glance. Any chance they are...


Q: 

What is a anterior infarction?

A: Anterior infarction and its importance will help you as i have tried to summarize it from an article: The importance and urgency of reperfusion increase with the amount of myocardium at risk. Electrocardiography is an extremely useful tool for determining the location and size of an acute myocardial infarct, and every patient in the emergency room or coronary care unit deserves a physician who knows how to use electrocardiography to obtain that information1. In acute anterior-wall myocardial infarction, the amount of myocardium at risk corresponds to the number of precordial leads showing QRS and ST-segment changes and to the magnitude and...


Q: 

What Is Anteroseptal infarction?

A: An anteroseptal infarction is a heart problem where part of the heart muscle dies and scars due to poor blood supply. In this case, the tissue damage is centered around the anteroseptal wall, the area between the left and right ventricles. This can be dangerous for the patient. It is necessary to receive treatment to address the cause of the tissue death and prevent additional damage.. This condition usually occurs as a result of a partial blockage along one of the branches of the left anterior descending (LAD) artery. When the whole artery is blocked, it can cause an acute myocardial infarction, where more of the heart is deprived of blood. In the case of lesser blockages, a limited

Q: 

In acute MI can the glucose level rise to 1.5 times the upper limit of normal and if so how does this occur?

A: We found a number of papers that described glucose levels during myocardial infarction.  The most recent paper looked at glucose levels in the context of acute coronary syndrome [1].  This reports: “Hyperglycemia in the context of acute coronary syndrome (ACS) is a common observation, and existing data suggest that high glucose levels are associated with increased in-hospital mortality.” The actual conclusion of their paper was: “High blood glucose during ACS is an independent predictor of long-term mortality and is significantly correlated with prognostic biomarkers. Glucose levels during ACS may be an important addition to the risk stratification of patients...
Q: 

What Is acute Coronary Syndrome?

A: acute coronary syndrome is a constellation of symptoms which emerge when the supply of blood to the heart is restricted. When a patient presents with acute coronary syndrome, immediate intervention is required to determine what is causing the condition and address it before permanent damage occurs. A doctor can diagnose acute coronary syndrome with the assistance of an electrocardiogram (ECG) as well as bloodwork and a patient interview. The key symptom is chest pain, which may radiate along the jaw and left arm. Patients can also experience nausea, vomiting, lightheadedness, clammy skin, sweating, and pale skin. The condition is caused by acute myocardial ischemia,...
Q: 

What is a inferior infarction?

A: An inferior infarction refers to a heart attack (myocardial infarction - MI) involving the inferior and possibly the posterior wall of the heart. This area of the heart is supplied blood by the right coronary artery and sometimes a part of it is supplied by a small branch of the left coronary artery. If the right coronary artery becomes diseases with lipid laden plaques, and a plaque ruptures, it can cause an infarction in the inferior or posterior walls of the heart. This will frequently cause chest pain, nausea, vomiting, sweating, shortness of breath, and possibly syncope. Sometimes, the only symptoms are nausea and vomiting, especially in women, the elderly, and those with diabetes...


Q: 

Does a heart muscle repair after a heart attack and at what rate?

A: acute myocardial infarction (heart attack) may result in death due to pump failure or ventricular fibrillation. If the patient survives, the infarct undergoes organization and healing. (Resorption of muscle --> thin wall + mural thrombi). During the first 2 to 3 weeks, the necrotic myocardium (dead cardiac muscle) is gradually replaced by granulation tissue; during the next 2 to 3 months, the granulation tissue is converted into fibrous scar. During healing, the thinned infarcted wall may expand to form a ventricular aneurysm (ballooning of the heart). Mural thrombi can form over the infarct and give rise to systolic emboli. If a person survives a heart attack, the heart muscle may return to normal or become a...


 
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